Inflammation Stocks List

Recent Signals

Date Stock Signal Type
2020-01-28 ATBPF Non-ADX 1,2,3,4 Bullish Bullish Swing Setup
2020-01-28 ATBPF Fell Below 20 DMA Bearish
2020-01-28 ATHJF Narrow Range Bar Range Contraction
2020-01-28 ENDV Upper Bollinger Band Walk Strength
2020-01-28 ENDV New 52 Week Closing High Bullish
2020-01-28 ENDV Shooting Star Candlestick Bearish
2020-01-28 ENDV Wide Range Bar Range Expansion
2020-01-28 ENDV New 52 Week High Strength
2020-01-28 ENDV Expansion Breakout Bullish Swing Setup
2020-01-28 ETST Non-ADX 1,2,3,4 Bearish Bearish Swing Setup
2020-01-28 ETST Narrow Range Bar Range Contraction
2020-01-28 ETST Calm After Storm Range Contraction
2020-01-28 MFST Bollinger Band Squeeze Range Contraction
2020-01-28 MFST Narrow Range Bar Range Contraction
2020-01-28 MFST Volume Surge Other
2020-01-28 MFST Fell Below 20 DMA Bearish
2020-01-28 MFST Expansion Breakdown Bearish Swing Setup
2020-01-28 TLTFF Stochastic Reached Oversold Weakness
2020-01-28 TLTFF Lizard Bullish Bullish Day Trade Setup
2020-01-28 TLTFF Hammer Candlestick Bullish
2020-01-28 TLTFF 50 DMA Support Bullish
2020-01-28 TLTFF Non-ADX 1,2,3,4 Bullish Bullish Swing Setup

Inflammation (from Latin: inflammatio) is part of the complex biological response of body tissues to harmful stimuli, such as pathogens, damaged cells, or irritants, and is a protective response involving immune cells, blood vessels, and molecular mediators. The function of inflammation is to eliminate the initial cause of cell injury, clear out necrotic cells and tissues damaged from the original insult and the inflammatory process, and initiate tissue repair.
The five classical signs of inflammation are heat, pain, redness, swelling, and loss of function (Latin calor, dolor, rubor, tumor, and functio laesa).[1] Inflammation is a generic response, and therefore it is considered as a mechanism of innate immunity, as compared to adaptive immunity, which is specific for each pathogen. Too little inflammation could lead to progressive tissue destruction by the harmful stimulus (e.g. bacteria) and compromise the survival of the organism. In contrast, chronic inflammation may lead to a host of diseases, such as hay fever, periodontitis, atherosclerosis, rheumatoid arthritis, and even cancer (e.g., gallbladder carcinoma). Inflammation is therefore normally closely regulated by the body.
Inflammation can be classified as either acute or chronic. Acute inflammation is the initial response of the body to harmful stimuli and is achieved by the increased movement of plasma and leukocytes (especially granulocytes) from the blood into the injured tissues. A series of biochemical events propagates and matures the inflammatory response, involving the local vascular system, the immune system, and various cells within the injured tissue. Prolonged inflammation, known as chronic inflammation, leads to a progressive shift in the type of cells present at the site of inflammation, such as mononuclear cells, and is characterized by simultaneous destruction and healing of the tissue from the inflammatory process.
Inflammation is not a synonym for infection. Infection describes the interaction between the action of microbial invasion and the reaction of the body's inflammatory response—the two components are considered together when discussing an infection, and the word is used to imply a microbial invasive cause for the observed inflammatory reaction. Inflammation on the other hand describes purely the body's immunovascular response, whatever the cause may be. But because of how often the two are correlated, words ending in the suffix -itis (which refers to inflammation) are sometimes informally described as referring to infection. For example, the word urethritis strictly means only "urethral inflammation", but clinical health care providers usually discuss urethritis as a urethral infection because urethral microbial invasion is the most common cause of urethritis.
It is useful to differentiate inflammation and infection because there are typical situations in pathology and medical diagnosis where inflammation is not driven by microbial invasion – for example, atherosclerosis, trauma, ischemia, and autoimmune diseases including type III hypersensitivity. Conversely, there is pathology where microbial invasion does not cause the classic inflammatory response – for example, parasitosis or eosinophilia.

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